LoopWHI ’26

Self vs Dependent Health Load

biomarkers
11,427 annual-checkup reports, 12-month window ending Dec 2025
claims
74,339 claims, ₹320.6 Cr incurred, latest 12-month window

Key takeaways

  • Dependents who got a checkup show glucose dysfunction at 51.4% against 31.3% for employees — but once you hold age constant, almost the entire gap disappears. The dependent biomarker pool is older, not metabolically different at the same age.
  • Anemia is the exception that survives the age control. Dependents run 14 to 20 percentage points more anemic than employees in every working-age band — a gap age cannot explain, and the cleanest age-independent dependent disadvantage in the biomarker file.
  • On three markers the employee is the sicker one. Low HDL, low B12, and raised liver enzymes are all higher in the working-age employee than in the dependent pool. "Dependents are uniformly sicker" is not what the biomarker data says.
  • Dependents drive 66.5% of claims and ₹217.4 Cr of incurred cost — more than twice the employee's ₹103.1 Cr — with the parent layer alone at ₹118.2 Cr, the single largest block in the policy.
  • The dependent layer is where screening reaches least and cost lands hardest. The biomarker file is overwhelmingly employees; dependents who do get tested are older, sicker on the markers age drives, and already carrying conditions the screening would have caught earlier.

Loop tests employees far more often than it tests the people on their policies. Of the annual-checkup reports with a usable HbA1c result, roughly 63% are the employee's own and 37% belong to a spouse, child, or parent — even though the claims book shows dependents are two-thirds of who the policy actually pays for. The dependent layer is under-screened and over-claimed at the same time. That asymmetry is the spine of this page: the biomarker file is a window into a population the policy mostly does not look at until it ends up in a hospital.

The headline numbers invite a simple reading. Dependents who do get a blood panel look much worse than employees on the marquee metabolic marker — 51.4% in the glucose-dysfunction band against 31.3%. The simple reading is that dependents are sicker. The data supports a sharper, more useful version of that claim, and corrects part of it.

The dependent biomarker pool is older, and on glucose that is almost the whole story#

Of dependents Loop ran an HbA1c on, 51.4% sit in the glucose-dysfunction band against 31.3% of employees — a 20-point gap. The instinct is to read that as a metabolic difference between the two groups. It is mostly an age difference between them.

Group n (HbA1c) Glucose dysfunction Diabetic-range HbA1c
Self (employee) 4,482 31.3% 8.6%
Dependent / other 2,606 51.4% 21.4%

Dependents who get tested look 20 points worse on glucose

Glucose dysfunction and diabetic-range HbA1c, employee vs dependent

self n = 4,482 · dependent n = 2,606
Self (employee)Dependent / other

Read with the age-controlled chart below: the dependent pool tested skews two decades older, and once age is held constant almost the entire gap disappears. The raw gap overstates the real one.

The dependent pool that shows up for a blood test is not a cross-section of dependents. It is heavily weighted toward parents — the layer most likely to be sent for a checkup precisely because someone is already worried about them.

Age band Employee reports Dependent reports
Under 20 0.1% 2.5%
20–29 37.9% 16.2%
30–39 42.7% 26.2%
40–49 14.3% 12.0%
50–59 3.8% 23.0%
60+ 1.1% 20.1%

The dependent pool tested is two decades older

Age mix of HbA1c reports — share of each group's reports by age band

Under 2020–2930–3940–4950–5960+
Employee reports
mostly 20s and 30s
Dependent reports
large 50-plus block
Values in %

Over 43% of dependents tested are 50 or older, against under 5% of employees. The dependent pool that shows up for a blood test is heavily weighted toward parents.

Hold age constant and the glucose gap collapses.

Age band Self dysfunction Dependent dysfunction Gap
20–29 16.2% 18.9% +2.7
30–39 33.4% 32.0% −1.4
40–49 51.4% 55.8% +4.4
50–59 65.5% 70.1% +4.6
60+ 65.3% 79.5% +14.2

Hold age constant and the glucose gap collapses

Glucose dysfunction by age band, self vs dependent

Self (employee)Dependent / other

The near-overlap is the finding: the 20-point headline gap is an age-mix effect, not a same-age metabolic difference. Only at 60+ does a real same-age dependent excess open up (+14.2 pp).

Through the working-age bands the two lines run together. A dependent in their 30s is no more likely to show glucose dysfunction than an employee in their 30s. The headline 20-point gap exists because the dependent group tested skews two decades older. Only at 60-plus does a real same-age dependent excess open up, and that band is small for employees by construction. The Blood Sugar page reads the dependent layer's 51.4% from the metabolic side, where it anchors the report's dependent-risk thesis.

Anemia is the one dependent disadvantage that age cannot explain#

Glucose equalises within age band. Anemia does not. Dependents run more anemic than employees at every working-age band, by a margin that holds whether the dependent is 25 or 55.

Age band Self anemic Dependent anemic Gap
20–29 13.5% 28.1% +14.6
30–39 14.9% 29.6% +14.7
40–49 13.9% 34.3% +20.4
50–59 17.9% 32.1% +14.1
60+ 31.9% 37.7% +5.8

Anemia is the dependent disadvantage age cannot explain

Anemia rate by age band, self vs dependent — paired dots, gap shown per band

Self (employee)Dependent / other
20–29
13.5% / 28.1%
30–39
14.9% / 29.6%
40–49
13.9% / 34.3%
50–59
17.9% / 32.1%
60+
31.9% / 37.7%
Self (employee) versus Dependent / other, values in %

Unlike the glucose chart, the two series run clearly apart across every working-age band (+14 to +20 pp), converging only at 60+. The contrast between this chart and the glucose one is the page's central argument.

The gender split rules out the obvious confound. Maternal-age iron loss would predict a female-driven gap; instead the dependent excess is sharpest among men.

Gender Self anemic Dependent anemic Gap
Female 31.9% 37.2% +5.3
Male 5.5% 21.7% +16.2

Dependent men are anemic at four times the rate of employee men

Anemia rate by gender and relationship to the policy

Self — female
31.9%
Dependent — female
+5.3 pp
37.2%
Self — male
5.5%
Dependent — male
+16.2 pp
21.7%
Values in %

5.5% vs 21.7% — a +16.2 pp gap, the largest in the cut. Maternal-age iron loss would predict a female-driven gap; instead the dependent excess is sharpest among men.

A male employee in this dataset is anemic 5.5% of the time. A male dependent is anemic 21.7% of the time — close to four times as often. Age does not explain it; the table above holds age flat and the gap persists. Gender does not explain it; the gap is largest within men. What is left is the difference between a population that gets an annual checkup and one that mostly does not. The Anemia page carries the full marker; this is the one dependent disadvantage that survives every control the biomarker file allows.

On three markers, the employee is the sicker one#

The thesis that dependents are uniformly worse off does not survive contact with the full marker set. On three of the seven markers tested, the working-age employee is the one in trouble.

Marker Self (employee) Dependent / other Who is worse
Glucose dysfunction 31.3% 51.4% Dependent (age-driven)
Diabetic-range HbA1c 8.6% 21.4% Dependent (age-driven)
Anemia 14.5% 32.1% Dependent (age-independent)
Low HDL (<40) 47.3% 37.6% Employee
Low B12 73.9% 61.9% Employee
Raised liver enzymes (SGPT) 20.9% 12.1% Employee
Low Vitamin D 82.9% 78.6% Roughly equal

On three markers, the employee is the sicker one

Seven biomarkers, self vs dependent — whichever dot sits further right is the worse-off group

Self (employee)Dependent / other
Glucose dysfunction
31.3% / 51.4%
Diabetic-range HbA1c
8.6% / 21.4%
Anemia
14.5% / 32.1%
Low HDL (<40)
47.3% / 37.6%
Low B12
73.9% / 61.9%
Raised liver enzymes (SGPT)
20.9% / 12.1%
Low Vitamin D
82.9% / 78.6%
Self (employee) versus Dependent / other, values in %

Anemia and the two glucose markers run against the dependent (glucose age-driven, anemia age-independent); low HDL, low B12, and raised SGPT all peak in the working-age employee. Vitamin D is roughly equal. 'Dependents are simply sicker' is not what the biomarker data says.

Low HDL, low B12, and raised liver enzymes all peak in the working-age employee. Age-controlled, the pattern sharpens: in the 30s, employees show low HDL at 52.1% against dependents' 41.0%, and raised liver enzymes at 23.7% against 12.9%. These are the markers of the desk-bound urban professional — the sedentary day, the late dinners, the drink after work, the vegetarian diet thin on B12. They are conditions the employee's own age band produces, not conditions the dependent layer is spared by youth.

Dependents are two-thirds of who the policy pays — and more than twice the cost#

The biomarker file looks at employees. The claims file is paid for by everyone else. Where Loop tests employees roughly two to one over dependents, the claims book runs the other way: two-thirds of what the policy pays out is for people other than the employee.

Relationship Share of claims Share of incurred Total incurred Average per claim
Employee 33.5% 32.2% ₹103.1 Cr ₹41,432
Spouse 22.7% 21.5% ₹69.0 Cr ₹40,849
Child 14.3% 9.4% ₹30.3 Cr ₹28,419
Parent / parent-in-law 29.4% 36.9% ₹118.2 Cr ₹53,981
All dependents 66.5% 67.8% ₹217.4 Cr ₹43,985

Dependents are two-thirds of who the policy pays

Share of claims and share of incurred cost by relationship — employee block vs the combined dependent block

74,339 claims · ₹320.6 Cr
EmployeeSpouseChildParent / parent-in-law
Share of claims
all dependents 66.5%
Share of incurred
all dependents 67.8%
Values in %

All dependents: 66.5% of claims, 67.8% of incurred — ₹217.4 Cr against the employee's ₹103.1 Cr. The parent layer alone (₹118.2 Cr, avg ₹53,981/claim) exceeds the entire employee block. The biomarker file Loop measures is mostly employee; the claims book it pays is mostly dependent. The two files are not linked at the individual level.

The dependent layer costs the policy ₹217.4 Cr against the employee's ₹103.1 Cr — more than two to one. The parent layer alone, at ₹118.2 Cr, exceeds the entire employee block. And the parent claim is the most expensive on average, at ₹53,981, because it concentrates in the cardiac admissions, fracture surgeries, and oncology that arrive in the 60s and 70s — the same age band where the glucose curve finally separates self from dependent.

The two datasets meet at the same conclusion from opposite directions. The biomarker file shows a dependent population that is under-tested and, where tested, carries an age-independent anemia burden and an age-driven metabolic one. The claims file shows that same population absorbing two-thirds of the bill, concentrated in conditions that build quietly for years before they are admitted. The screening gap and the cost concentration are the same fact seen at two ends of the disease timeline.

For the deeper claims-side breakdown of the dependent layer — spouse, child, and parent as clinically distinct populations, with the gender and account-tier cuts — see Who the Policy Pays For. The Parent Claims page goes further into the costliest sub-layer, the ₹118.2 Cr the policy spends on the workforce's parents. This page's contribution is the biomarker half: the evidence that the dependent cost is foreshadowed in screening data the policy already could be collecting.

The dependent layer is the cleanest preventive-investment case in the dataset#

Three facts the 2026 data establishes together:

  • The dependent layer is screened far less than the employee — roughly one dependent biomarker report for every two employee reports, against a claims book that is two-thirds dependent.
  • Where dependents are screened, the one disadvantage that age cannot explain is anemia — a slow, correctable, screening-sensitive condition that runs four times higher in dependent men than employee men.
  • The dependent layer absorbs ₹217.4 Cr of incurred cost, more than twice the employee's, concentrated in late-life conditions that announce themselves on a blood panel long before they reach a hospital.

References

  1. 1Anand K, Shah B, Yadav K, et al. Are the urban poor vulnerable to non-communicable diseases? A survey of risk factors for non-communicable diseases in urban slums of Faridabad. National Medical Journal of India, 2007;20(3):115–120.
  2. 2Didzun O, De Neve JW, Awasthi A, et al. Anaemia among men in India: a nationally representative cross-sectional study. The Lancet Global Health, 2019;7(12):e1685–e1694. https://doi.org/10.1016/S2214-109X(19)30440-1
  3. 3Anjana RM, Unnikrishnan R, Deepa M, et al. Metabolic non-communicable disease health report of India: the ICMR-INDIAB national cross-sectional study. The Lancet Diabetes & Endocrinology, 2023;11(7):474–489. https://doi.org/10.1016/S2213-8587(23)00119-5
  4. 4Green R, Allen LH, Bjørke-Monsen AL, et al. Vitamin B12 deficiency. Nature Reviews Disease Primers, 2017;3:17040. https://doi.org/10.1038/nrdp.2017.40